tDCS stimuli change the memory of pain acute pain is treated with analgesics, according to WHO stages schema first with peripheral effective, later with weak and highest expression with strong opioids. The pain afferent, the “Inbox”area is influenced so to speak. The pharmacological range under the notion that a Molesta through pain not only by increased delivery of signal from the periphery, but also by weakening the efferent pain inhibition is conceivable extends for chronic pain. Finally it applies to deviate from the Descartes’schen thinking of a sole pain afferent and the no longer new knowledge to take into account that our perception of pain ultimately results from the balance of pain afferent minus the pulses filtered away by the efferent pain inhibition. The efferent pain inhibition can be improved by the use of serotonin – and norepinephrine-enriching drug, also by using CA channel modulators and NA channel blockers.
This is pain therapy already banal and one like it already hardly repeat, that the use of antidepressants and anticonvulsants pain therapeutic treatment is one of the Basic program. Anyone who knows the reality of primary medical care, know that is that not much has gotten around. Pain therapists explain the stubborn persistence of painful after occurring chronicity with the emergence of pain memory. The pain memory the result of Neuroplatizitat is neurophysiologisch, functional and micro-structurally alter the ability of the central nervous system, in the context of learning and to adapt. Neuro-plastic operations benefit us to the advantage, when it comes to learn repeated experiences and getting better and faster. Everyone is familiar with the examples of the violinist, whose brain representation for violin playing is increasing with increasing expertise and brings more and more in the fMRI fields “to the light”. There are also neuro plastic operations, to our detriment run maladaptive neuroplasticity. The memory of the pain is such a faulty construction due to neuroplasticity.
